Information om | Engelska ordet ANTIPHOSPHOLIPID


ANTIPHOSPHOLIPID

Antal bokstäver

16

Är palindrom

Nej

28
AN
ANT
HO
HOL
HOS

1

1

AD
ADH
ADI


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Exempel på hur man kan använda ANTIPHOSPHOLIPID i en mening

  • Antiphospholipid syndrome, or antiphospholipid antibody syndrome (APS or APLS), is an autoimmune, hypercoagulable state caused by antiphospholipid antibodies.
  • hemorrhage from sepsis, adrenal vein thrombosis, hypercoagulable states such as heparin-induced thrombocytopenia and antiphospholipid syndrome.
  • Libman–Sacks endocarditis is a form of non-bacterial endocarditis that is seen in association with systemic lupus erythematosus, antiphospholipid syndrome, and malignancies.
  • To rule out other causes for hypercoagulation, it may be appropriate to check the antithrombin, protein C, protein S, factor V Leiden, prothrombin G20210A, and antiphospholipid antibodies.
  • Other causative conditions include infections, toxicities, antiphospholipid syndrome, cryoglobulinemia, neoplasms.
  • Arteriosclerosis obliterans has been postulated as the cause, along with errors of the clotting and fibrinolytic pathways such as antiphospholipid syndrome.
  • Antibodies directed against annexin A5 are found in patients with a disease called the antiphospholipid syndrome (APS), a thrombophilic disease associated with autoantibodies against phospholipid compounds.
  • Hypercoagulability states as a pre-existing condition in pregnancy include both acquired ones, such as antiphospholipid antibodies, and congenital ones, including factor V Leiden, prothrombin mutation, proteins C and S deficiencies, and antithrombin III deficiency.
  • Splinter hemorrhages are not specific to any particular condition, and can be associated with subacute infective endocarditis, scleroderma, trichinosis, systemic lupus erythematosus (SLE), rheumatoid arthritis, psoriatic nails, antiphospholipid syndrome, haematological malignancy, and trauma.
  • In addition to the previously noted vascular occlusions, development of different thromboembolic manifestations simultaneously or within one or two weeks must occur, and the patient must have an underlying inherited or acquired hypercoagulable state (other than antiphospholipid syndrome).
  • She has a specialist clinical and research interest in venous thromboembolism, thromboprophylaxis, antiphospholipid syndrome, lupus erythematosus, and obstetric haematology.
  • USS can present similar to the following diseases, which have to be excluded: fulminant infections, disseminated intravascular coagulation, autoimmune hemolytic anemia, Evans syndrome, the typical and atypical form of hemolytic uremic syndrome, HELLP (hemolysis, elevated liver enzymes, low platelets) syndrome, pre-eclampsia, heparin-induced thrombocytopenia, cancer that is often accompanied with metastasis, kidney injury, antiphospholipid antibody syndrome, and side effects from hematopoietic stem cell transplantation.
  • Autoantibodies may be present including antinuclear, antiphospholipid, and anticardiolipin antibodies.
  • Some thrombotic and coagulopathic disorders are disseminated intravascular coagulation (DIC), hypercoagulable states (like antiphospholipid antibodies), thrombotic thrombocytopenic purpura (TTP), warfarin-induced skin necrosis, heparin-induced thrombocytopenia, and paroxysmal nocturnal hemoglobinuria.


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