Anagram & Information om | Engelska ordet CATECHOLAMINES

Exempel på hur man kan använda CATECHOLAMINES i en mening

• Beta blockers are competitive antagonists that block the receptor sites for the endogenous catecholamines epinephrine (adrenaline) and norepinephrine (noradrenaline) on adrenergic beta receptors, of the sympathetic nervous system, which mediates the fight-or-flight response.
• It is the innermost part of the adrenal gland, consisting of chromaffin cells that secrete catecholamines, including epinephrine (adrenaline), norepinephrine (noradrenaline), and a small amount of dopamine, in response to stimulation by sympathetic preganglionic neurons.
• Clonidine works by slowing the pulse rate and exerts a reduction of serum concentrations of renin, aldosterone, and catecholamines.
• -DOPA is the precursor to the neurotransmitters dopamine, norepinephrine (noradrenaline), and epinephrine (adrenaline), which are collectively known as catecholamines.
• These drugs can act through several mechanisms, such as directly activating postsynaptic receptors, blocking breakdown and reuptake of certain neurotransmitters, or stimulating production and release of catecholamines.
• When a distinction is made, the "aortic bodies" are chemoreceptors which regulate the circulatory system, while the "paraaortic bodies" are the chromaffin cells which manufacture catecholamines.
• The antihypertensive actions of reserpine are largely due to its antinoradrenergic effects, which are a result of its ability to deplete catecholamines (among other monoamine neurotransmitters) from peripheral sympathetic nerve endings.
• Expression of the Gly-16 allele (glycine at position 16) results in greater receptor downregulation by endogenous catecholamines at baseline compared to Arg-16.
• Another possibility is that alcohol consumption increases the level of catecholamines, which increased the level of P-waves, and therefore the risk of arrhythmia.
• Reduction in catecholamines and their metabolites (normetanephrine, metanephrine, and 4-hydroxy-3-methoxymandelic acid) result from the inhibition of tyrosine using AMPT.
• The painful, though rarely life-threatening sting involves the injection of a complex venom containing amines (histamine, tyramine, serotonin, catecholamines), peptides, and proteins, including many hydrolases.
• COMT eliminates biologically active catechols present in catecholamines (dopamine, norepinephrine, and epinephrine) and their hydroxylated metabolites.
• However, due to the hormone-producing function of the adrenal glands, some noncancerous adrenal tumors may produce too much hormones, such as aldosterone (called primary aldosteronism), cortisol (called Cushing's disease or Cushing's syndrome), or catecholamines (called pheochromocytoma).
• Catechol, for example, is synthesized from o-hydroxy and o-alkoxy phenyl aldehydes and ketones, and is used as the starting material for synthesis of several compounds, including the catecholamines, catecholamine derivatives, and 4-tert-butylcatechol, a common antioxidant and polymerization inhibitor.
• Indolamines are a classification of monoamine neurotransmitter, along with catecholamines and ethylamine derivatives.
• When a distinction is made, the "aortic bodies" are chemoreceptors which regulate circulation, while the "paraaortic bodies" are the chromaffin cells which manufacture catecholamines.
• The celiac ganglion is part of the sympathetic prevertebral chain possessing a great variety of specific receptors and neurotransmitters such as catecholamines, neuropeptides, and nitric oxide and constitutes a modulation center in the pathway of the afferent and efferent fibers between the central nervous system and the ovary.
• Whereas both types of allostasis are associated with increased release of cortisol and catecholamines, they differentially affect thyroid homeostasis: Concentrations of the thyroid hormone triiodothyronine are decreased in type 1 allostasis, but elevated in type 2 allostasis.
• Guanethidine is an antihypertensive drug that reduces the release of catecholamines, such as norepinephrine.
• These mechanisms depend on current synaptic "state", as set by ongoing extrinsic influences such as the level of synaptic inhibition, the activity of modulatory afferents such as catecholamines, and the pool of hormones affecting the synapses under study.

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